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TGF-β1对糖尿病肾病早期ECM沉积影响的实验研究(3)
http://www.100md.com 2008年2月24日 《中国实用医药》 2007年第31期
     3 Phillips AO,Steadman R,Diabetic nephropathy:the central role of renal proximal tubular cells in tubulointerstitial injury.Histol Histopathol,2002,17(1):247-252.

    4 Nangatu M,Mechanisms of tubulointerstitial injury in the kidney:final common pathways to end stage renal failure.Intern Med,2004,43(1):9-17.

    5 Weigert C,Brodbeck K,Brosius FC 3rd,et al.Evidence for a novel TGF-betal-independent mechanism of fibronectin production in mesangial cells over expressing glucose transporters.Diabetes,2003,52:527-535.

    6 李振江,雷洁,钟芳红,等.糖尿病肾病TGF-β1,IV型胶原水平变化的临床意义及辛伐他汀的治疗作用.第四军医大学学报,2004,25(10):942-944.

    7 Chen S,Jim B,Ziyadeh FN.Diabetic nephropathy and TGF-beta:transforming our view of glomerulosclerosis and fibrosis build up.Semin Nephrol,2003,23(6):532-543.

    8 Nicholas SB.Advances in pathogenetic mechanisms of diabetic nephropathy.Cell Mol Biol,2003,49(8):1319-1325.

    9 Li JH,Zhu HJ,Huang XR,et al.Smad 7 inhibits fibrotic effect of TGF-beta on renal tubular epithelial cells by blocking smad 2 activaiton.J Am Soc Nephrol,2002,13(6):1464-1472.

    10 Guh JY,Chuang TD,Chen HC,et al.Beta-hydroxybutyrate-induced growth inhibition and collagen production in HK-2 cells are dependent on TGF-beta and Smad 3.Kidney Int,2003,64(6):2041-2051., http://www.100md.com(苏 宁 罗荣敬 邝枣园 邢婷文 何彦丽 朱锦锦 陈晓瑜)
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